Scopus h-index:35
Article:77
Review Article:8
Comment/Debate:2
Chapter:1
Selected Publications
- Author Correction: Maladaptive positive feedback production of ChREBPβ underlies glucotoxic β-cell failure (Nature Communications, (2022), 13, 1, (4423), 10.1038/s41467-022-32162-x)
- Maladaptive positive feedback production of ChREBPβ underlies glucotoxic β-cell failure
- T3 and glucose increase expression of phosphoenolpyruvate carboxykinase (PCK1) leading to increased β-cell proliferation
- Regulation of Pdx1 by oxidative stress and Nrf2 in pancreatic beta-cells
- Disrupting the DREAM complex enables proliferation of adult human pancreatic β cells
- Nrf2 Regulates b-Cell Mass by Suppressing b-Cell Death and Promoting b-Cell Proliferation
- The c-Myc Oncogene Maintains Corneal Epithelial Architecture at Homeostasis, Modulates p63 Expression, and Enhances Proliferation During Tissue Repair
- Pharmacological blockade of the EP3 prostaglandin E2 receptor in the setting of type 2 diabetes enhances β-cell proliferation and identity and relieves oxidative damage
- Adaptive and maladaptive roles for ChREBP in the liver and pancreatic islets
- The many lives of Myc in the pancreatic β-cell